Golf Lessons at Torrey Pines with Michael Major, PGAI’ve been teaching golfers from beginners to touring professionals, collegiate players to accomplished amateurs and our local junior to Hollywood celebrities. I have taught thousands of lessons and helped all levels of golfers build, grow and improve their game of golf for over 2. Torrey Pines including the full swing, shortgame, putting, course management and the mental side of the game. Customized programs are offered to get you hitting the ball straighter and farther with fewer strokes, more understanding, less effort and added enjoyment. Call me on my cell at 6. I make a promise to you to do my best to improve your overall game. Click here to check out my Bio. Lesson Services Offered: Lesson Features: All inclusive customized packages including advanced reservations, green fees, instruction, cart, range balls & choice of Torrey Pines North or South Course with any Playing Lessons or Golf Schools. Tee times with no advanced reservation fee on Playing Lessons, Prepare and Play Package or Golf Schools. Unlimited range balls on Private Lessons and Golf Schools. High tech computer video analysis with Private Lessons, P& P Packages and Golf Schools. Unsurpassed PGA golf instruction with over 2. Torrey Pines. For all levels: Beginners: If you’re a beginner, whether your female, male, junior, senior, or have a physical handicap, my services will help you achieve a level of comfort with additional knowledge, awareness and understanding. When either playing on a course, practicing at a range or anywhere on the golf facility the intimidation factor will be put at ease. Intermediates: As an intermediate, you will enhance your knowledge and start understanding what your club does to the ball and what your swing does to the club. Transform aggravation and frustration into knowledge and understanding, so you enjoy your game. Accomplished Players: As for the accomplished player, fine- tune your skills and incorporate a physical and mental proficiency to elevate your game to the next level.” I was fortunate enough to have my oldest daughter purchase a 6 lesson private instruction plan from Michael Major at Torrey Pines. Acetone (EHC 2. 07, 1. INTERNATIONAL PROGRAMME ON CHEMICAL SAFETY. ENVIRONMENTAL HEALTH CRITERIA 2. ACETONE. This report contains the collective views of an international group of. Important For All: Emotional and Social Support. The Stress And Dangers Of Antidepressant Drugs And Avoiding Them With Subtle Energies. As emphasized in several. United Nations Environment Programme, the International. Labour Organisation, or the World Health Organization. Environmental exposure. I. International Programme on Chemical Safety II. Series. ISBN 9. 2 4 1. NLM Classification: QD 3. K2). ISSN 0. 25. 0- 8. X. The World Health Organization welcomes requests for permission to. Errors and omissions excepted, the names of. Uses and sources of exposure. Uses and emissions into the environment. Environmental transport, distribution and transformation. The Boeing B-52 Stratofortress is an American long-range, subsonic, jet-powered strategic bomber. The B-52 was designed and built by Boeing, which has continued to. 03/30/2017 Boynton Briefing - March 30, 2017. 17-017656 2206 hours Burglary to Residence 436 SW 7th Ct. Units responded to the above address in reference to a. Today we are the market leader and one of the largest independent transporters and. Court of Appeals of Virginia Published Opinions. These opinions are available as Adobe Acrobat PDF documents. The Adobe Acrobat Viewer (free from Adobe) allows you to. Environmental levels and human exposure. Kinetics and metabolism. Effects on laboratory mammals and in vitro systems. Effects on other organisms in the laboratory and field. IDENTITY, PHYSICAL AND CHEMICAL PROPERTIES, AND ANALYTICAL. METHODS. 2. 1. Physical and chemical properties. Physical properties. Chemical properties. Conversion factors. Analytical methods. Biological media. SOURCES OF HUMAN AND ENVIRONMENTAL EXPOSURE3. Natural occurrence. Anthropogenic sources. UPDATE: The Law and Legal Research in Swaziland. By Buhle Dube and Alfred Magagula. Update by Alfred Magagula and Sibusiso Nhlabatsi. Alfred Sgcibelo Magagula is a. Simmons was born in the Melbourne suburb of Fitzroy, to American father Dave and Australian mother Julie. When his parents met in 1991, Julie was a. Production levels and processes. Releases. 3. 2. 3. Air. 3. 2. 3. 2 Water. Soil. 4. ENVIRONMENTAL TRANSPORT, DISTRIBUTION AND TRANSFORMATION4. Transport and distribution among media. Biotransformation. Finding The Right Contractor Shouldn't Be Painful Doing the right home improvement or remodeling project can add real value to any type of home, if done correctly and. Online legal research service for legal and law related materials and services, including searches of United States and international legal materials, journals. Bioconcentration and biomagnification. Biodegradation. 4. Microbial degradation. Bioavailability from environmental media. Interaction with other physical, chemical or biological. Ultimate fate following use. ENVIRONMENTAL LEVELS AND HUMAN EXPOSURE5. Environmental levels. Air. 5. 1. 1. 1 Indoor air. Soil and sediment. Other environmental levels. General population exposure. Occupational exposure. KINETICS AND METABOLISM IN LABORATORY ANIMALS AND HUMANS6. Inhalation exposure. Human studies. 6. Experimental animal studies. Oral exposure. 6. Human studies. 6. Experimental animal studies. Dermal exposure. 6. Human studies. 6. Experimental animal studies. Absorption summary. Inhalation exposure. Human studies. 6. Experimental animal studies. Injection exposure. Distribution summary. Experimental animal studies. Metabolism summary. Elimination and excretion. Human studies. 6. Occupational exposure studies. Experimental animal studies. Elimination/excretion summary. Physiologically based pharmacokinetic model. Retention and turnover. EFFECTS ON LABORATORY MAMMALS AND IN VITRO TEST SYSTEMS7. Short- term toxicity. Skin and eye irritation. Longer- term toxicity. Reproductive toxicity, embryotoxicity and teratogenicity. Factors modifying toxicity; toxicity of metabolites. Mechanisms of toxicity - mode of action. Effects on humans. Non- occupational exposure. Occupational exposure. Subpopulations at special risk. EFFECT ON OTHER ORGANISMS IN THE LABORATORY AND FIELD9. Aquatic organisms. Acute toxic effects on aquatic fauna. Chronic effects on aquatic fauna. Effects on aquatic plants. Effects on bacteria and protozoa. Terrestrial organisms. Effects on fauna. EVALUATION OF HUMAN HEALTH RISKS AND EFFECTS ON THE ENVIRONMENT1. Evaluation of human health effects. Evaluation of effects on the environment. PREVIOUS EVALUATION BY INTERNATIONAL BODIES. PCS/9. 0. 6. 9, Geneva, World. Health Organization). The selection of chemicals has been based on the. Observers do not. Task Group members. Such a procedure ensures the transparency and. Anderson, British Industrial Biological Research Association. BIBRA) Toxicology International, Carshalton, Surrey, United Kingdom. Dr Sin- Eng Chia, Department of Community, Occupational and Family. Medicine, National University of Singapore, Faculty of Medicine. Mr J. Fawell, National Centre for Environmental Toxicology, Medmenham. United Kingdom. Dr L. Fishbein, Fairfax, Virginia, USA ( Chairman). Dr H. Hansen, Division of Toxicology, Agency for Toxic. Substances and Disease Registry, Atlanta, Georgia, USA. Mr H. Malcolm, Institute of Terrestrial Ecology, Monks Wood. Experimental Station, Huntingdon, United Kingdom ( Co- Rapporteur). Dr M. V. Park, Edinburgh Centre for Toxicology, Edinburgh, United. Mr D. J. Reisman, National Center for Environmental Assessment, US. Environmental Protection Agency, Cincinnati, Ohio, USA. Co- Rapporteur). Dr A. Wibbertman, Fraunhofer Institute for Toxicology and Aerosol. Research, Hanover, Germany ( Vice- Chairman). Dr D. Morgott, Toxicological Sciences Laboratory, Health, Safety and. Environment, Eastman Kodak Company, Rochester, New York, USA. American Industrial Health Council). Dr D. Owen, Shell Chemicals Europe Limited, London, United Kingdom. European Centre for Ecotoxicology and Toxicology of. Dr P. Montuschi, Department of Pharmacology, Catholic University of. Sacred Heart, Rome, Italy (representing the International Union of. Pharmacology). Secretariat. Dr E. Smith, International Programme on Chemical Safety, World Health. Organization, Geneva, Switzerland. WHO TASK GROUP ON ENVIRONMENTAL HEALTH CRITERIA FOR ACETONE. A WHO Task Group on Environmental Health Criteria for Acetone met. British Industrial Biological Research Association (BIBRA). Toxicology International, Carshalton, Surrey, United Kingdom, from 1. December 1. 99. 7. Jaggers opened the meeting and welcomed the. Smith, IPCS. welcomed the participants on behalf of the Director, IPCS, and the. IPCS cooperating organizations (UNEP/ILO/WHO). The Task Group. reviewed and revised the draft criteria monograph and made an. Reisman, US Environmental Protection Agency, Cincinnati. USA, prepared the first draft of this monograph. The second draft. IPCS Contact Points for Environmental Health Criteria. Mr. Jenkins, both of the IPCS Central Unit. SUMMARY1. 1 Properties. Acetone (relative molecular mass = 5. The explosive. limits in air are 2. It has a high evaporation rate. Hg at 2. 0. It is miscible with water and organic solvents. The cumene peroxidation. Major uses are in the production of methyl methacrylate. A, methyl. isobutyl ketone, drug and pharmaceutical applications, and as a. There are also food. Certain detergents/cleansers. The average. half- life for acetone degradation in the atmosphere is approximately. Acetone can be physically removed from air by wet deposition. Volatilization. of acetone from the aquatic environment can be a significant transport. Acetone is a volatile compound that will evaporate from dry. Since acetone is miscible in water, it can leach readily in. Concurrent biodegradation may diminish the general. Acetone also occurs as a metabolic component in blood, urine. It occurs as a biodegradation product of sewage. Acetone has been detected in a variety of plants and foods. Natural. emissions from a variety of tree species contain acetone vapour. Human. sources of emissions to the aquatic environment include waste- water. A major source of human emission to air is. Acetone is an emission product from the. It is also emitted in exhaust. Concentrations of acetone. It can be formed endogenously in the mammalian. Fasting, diabetes mellitus and. Under. normal conditions, the production of ketone bodies occurs almost. The process is continuous, and the three products are excreted. Two of these ketone. Acetone, in contrast, is non- ionic and is derived. Endogenous acetone is eliminated from the body either by. Under. normal circumstances, metabolism is the predominant route of. Studies of rats indicate. However, lower and higher respiratory. The nasal cavities of humans and. In mice, maximum acetone. Acetone. is rapidly cleared from the body by metabolism and excretion. Exhalation is the major route of. CO2) and the. fraction of administered acetone that is exhaled as unchanged acetone. Urinary excretion of acetone and its metabolites. CO2. Carbon from orally administered acetone. CO2 in. exhaled breath. Metabolically, acetone is degraded to acetate and. The first pathway proceeds through the initial catalytic. The second gluconeogenic pathway involves the formation of. The 4- h inhalation LC5. No consistent changes were noted in. In female rats. administered the highest concentration, 5. In addition, a 1. Female mice given. No. toxic effects were observed in male mice administered 2. Thirteen- week exposures to concentrations < 1. NOELs for female rats (1. Positive results are restricted to a single test. Acetone is not considered to be. An atmospheric. concentration of 5. NOAEL for. developmental toxicity in both mice and rats. In a gavage study. Negative results in vivo. No investigations of the effect of oral doses of acetone on. Acute exposures. of humans to atmospheric concentrations as high as approx. More. severe transient effects (including vomiting and fainting) were. Acute exposures to acetone have. Females exposed to atmospheric. There was inhibition of growth. Chlamydomonas eugametos exposed to acetone for 4. Photosynthesis was increased in Scendesmus quadricauda and. C. The diatom Nitzschia linearis also seemed very resistant. EC5. 0 of 1. 1 4. Similarly, the saltwater. Skeletonema costatum was very resistant with 5- day EC5. This may. be related to cell wall differences. Fertile mallard eggs were not affected when immersed in 1. White Leghorn chicken eggs injected with 5 . IDENTITY, PHYSICAL AND CHEMICAL PROPERTIES, AND ANALYTICAL METHODS2. Chemical identity. Chemical name acetone. Synonym(s) dimethyl ketone; 2- propanone. Chemical formula C3. H6. O. Chemical structure. O. . It is miscible with water and organic solvents such as ether. Nelson & Webb, 1. The physical. properties of acetone, such as high evaporation rate, low viscosity. Krasavage et. al., 1. The physical properties of acetone are shown in Table 1. The chemical reactivity of acetone is commercially. A and other derivatives (SRI, 1. Physical and chemical properties of acetone. Property Value/descriptiona Reference. Relative molecular mass 5. Riddick et al, (1. Colour Clear colourless Sax & Lewis (1. Physical state Liquid Sax & Lewis (1. Melting point - 9. Acetone is a well- studied chemical and is used frequently in. This section is a review of the more established and. Ben Simmons - Wikipedia. This article is about the Australian basketball player. For the British bobsleigher, see Ben Simons. Benjamin David Simmons (born 2. July 1. 99. 6) is an Australian professional basketball player for the Philadelphia 7. National Basketball Association (NBA). A versatile forward from Melbourne, Simmons attended Box Hill Senior Secondary College before moving to the United States to attend Montverde Academy in Montverde, Florida. He played one season of college basketball for Louisiana State University (LSU), where he was named a consensus first- team All- American and the USBWA National Freshman of the Year. Simmons was selected with the number one overall pick in the 2. NBA draft by the 7. Melbourne- born number one overall pick in 1. He has also represented the Australian national team. Early life. Simmons was born in the Melbourne suburb of Fitzroy, to American father Dave and Australian mother Julie. When his parents met in 1. Julie was a divorced single mother with four children. After their marriage in 1. Olivia and Ben to their family. Even though the six children have two different biological fathers, and their age span is 1. Melissa to Ben), they were raised as if they were full siblings. Simmons began playing basketball at the age of seven in the Newcastle Hunters' under- 1. Lake Macquarie and Newcastle. In grade 7, while attending Whitefriars College, Simmons was named MVP after he helped Whitefriars win the Year- 7 Division 1. A Basketball premiership. In April 2. 01. 3, he helped Montverde rally from a 1. New Jersey's St Benedict's 6. On the season, Simmons averaged 1. He recorded 8. 8 blocks as well. Montverde finished the season with a 2. Simmons had 2. 4 points and 1. MVP honours. In doing so, he became just the second player from a Florida high school to win the award behind Austin Rivers in 2. He again earned MVP honours after recording 5. No. 1 player in the nation in 2. Relishing the opportunity to show his teammates where it all began for him. His double- double of 2. Tigers, playing at the Newcastle Basketball Stadium where his father played professionally for the Newcastle Falcons in the late 1. His 4. 3 points were the most for a Tigers player since Shaquille O'Neal had 4. Northern Arizona on 2. December 1. 99. 1. He was also named to the eight- player All- SEC Freshman team. LSU bowed out of the tournament with a demoralising 7. He led the team in all major statistical categories except three- point and free throw shooting, finishing with averages of 1. On 2. 1 March 2. 01. Simmons declared for the 2. NBA draft, forgoing his final three years of college eligibility. After receiving an X- ray and MRI of the foot and ankle, it was determined that Simmons suffered a fracture of the fifth metatarsal bone of his right foot. He subsequently scored four total points in the two- game series. I will play for Australia again when the time is right. Retrieved 5 November 2. Retrieved 2. 4 November 2. Retrieved 2. 0 June 2. Retrieved 3. 0 November 2. Retrieved 2. 4 November 2. Retrieved 2. 8 January 2. Retrieved 2. 4 November 2. Whitefriars. vic. Retrieved 2. 0 September 2. Retrieved 1. 8 January 2. Retrieved 1. 7 November 2. Retrieved 1. 7 November 2. Retrieved 2. 5 November 2. Retrieved 2. 4 November 2. Retrieved 1. 7 November 2. Retrieved 2. 9 March 2. Retrieved 2. 4 November 2. Retrieved 1. 5 August 2. Retrieved 2. 4 November 2. Retrieved 2. 4 November 2. Retrieved 2. 0 August 2. Retrieved 2. 3 February 2. Retrieved 2. 3 February 2. Retrieved 1. 0 March 2. Retrieved 2. 0 March 2. Retrieved 2. 4 March 2. Retrieved 5 April 2. Retrieved 5 April 2. Retrieved 1. 2 April 2. Retrieved 2. 6 April 2. Retrieved 1. 8 August 2. Retrieved 1. 8 August 2. Retrieved 3. 1 August 2. Retrieved 3. 1 August 2. Retrieved 3. 1 August 2. Retrieved 2. 1 October 2. Retrieved 1. 1 November 2. Retrieved 1. 3 November 2. Retrieved 2. 4 November 2. Retrieved 3 December 2. Retrieved 6 January 2. Retrieved 1. 9 February 2. Retrieved 2. 1 February 2. Retrieved 1. 1 March 2. Retrieved 1. 3 March 2. Retrieved 1. 3 March 2. Retrieved 2. 1 March 2. Retrieved 2. 1 March 2. Retrieved 1. 7 March 2. Retrieved 2. 1 June 2. Retrieved 2. 1 June 2. Retrieved 2. 3 June 2. Retrieved 3 July 2. Retrieved 4 January 2. Retrieved 1. 0 July 2. Retrieved 1. 8 July 2. Retrieved 1. 8 July 2. Retrieved 3. 0 September 2. Retrieved 1. 6 October 2. Retrieved 2. 4 February 2. Retrieved 2. 4 February 2. Retrieved 8 April 2. Retrieved 2. 4 November 2. Retrieved 2. 4 November 2. Retrieved 2. 4 November 2. Retrieved 9 December 2. Retrieved 1. 0 April 2. Retrieved 1. 3 August 2. Retrieved 2. 0 June 2. Retrieved 2. 4 November 2. Retrieved 2. 6 June 2. Retrieved 2. 4 July 2. Retrieved 2. 4 July 2. Retrieved 3. 0 December 2. Stress in Adults, Youth and Seniors- keys to a healtly life“Out there things can happenand frequently do to people as brainy and footsy as you,And when things start to happen, don’t worry. Seuss. Men/Women Families Children Teens College Students Seniors Pets. Important For All: Emotional and Social Support. The Stress And Dangers Of Antidepressant Drugs And Avoiding Them With Subtle Energies. As emphasized in several Newsletters, antidepressants are not much better than placebos in the vast majority of patients and can have serious side effects. This lack of efficacy was again confirmed in a recent Journal of The American Medical Association article, which also reported that the newer serotonin reuptake inhibitors (SSRIs) offered no advantages over older drugs. And they have alarmingly high rates of suicide, homicide and aggressive behavioral complications that have been hidden or minimized in advertising that hypes their benefits. For example, a previously court sealed 2. Glaxo. Smith. Kline documents uncovered during recent litigation now leaves little doubt that the company had data from clinical trials demonstrating an increased risk of suicide as early as 1. Internal reports and memos reveal that the risk of suicidal behavior in adults increased eightfold in patients on Paxil. Over 3,5. 00 documented reports of suicide, self- mutilation, murder and other violent acts involving SSRIs are posted at http: //www. Such problems are so prevalent in younger individuals that Paxil and similar SSRIs are banned in the UK and elsewhere for children and teenagers. Their lack of efficacy is not surprising, since there is no solid evidence that depression is due to serotonin deficiency or disturbances in other neurotransmitters. Part of the problem is that depression is not a discrete diagnosis like diabetes, where the causes are clear and there is general agreement on objective diagnostic laboratory criteria. Depression is simply a description of certain symptoms that can have many causes, which explains why we have so many treatment options. These include: various classes of pharmaceuticals (monoamine oxidase inhibitors, tricyclic and tetracyclic antidepressants, selective serotonin reuptake inhibitors, lithium, estrogen and amphetamines); supplements (melatonin, St. Johns wort, Ginkgo biloba, Panax ginseng, SAM- E, fish oils, omega- 3, glutathione, calcium, vitamin D, folic acid and other B vitamins); psychiatric interventions (psychoanalysis, cognitive restructuring, group therapy, stress reduction); sleep deprivation; exercise; ultraviolet light; acupuncture; surgery (stereotactic cingulotomy, deep brain or vagal stimulation with an implanted device); electroconvulsive therapy (ECT); and novel bioelectromagnetic approaches such as cranioelectrical stimulation and repetitive transcranial magnetic stimulation. There is no other disorder for which so many very different therapies are available, without any certainty of which will be effective for any given patient. Depression can also be seen in thyroid and other endocrine disturbances, particularly in post partum and menopausal females, in Parkinson’s disease and stroke victims, as well as a side effect of many medications. One exception may be electroconvulsive (shock) therapy, which is beneficial in almost all severely depressed individuals. Although it has been utilized for more than eighty years and is currently administered to over one million patients annually, we still don’t know why it works. However, it has stimulated interest in other electrotherapeutic approaches, the latest being simulation of the habenula, a tiny nerve structure found deep in both sides of the brain. Studies have shown that the habenula is hyperactive in depression and that this can be suppressed by stereotactically placing electrodes on each habenula connected with wires under the skin to an electronic impulse generator implanted in the chest, which can be controlled with an external device. Researchers just reported complete remission in a 6. ECT. The success of the procedure was confirmed when the patient had a bicycle accident which required surgery, and a preoperative ECG had to be obtained. The brain pacemaker was accidentally switched off for a few days, and the depression promptly returned. Following reactivation, the patient completely and quickly recovered. Repetitive transcranial magnetic stimulation (r. TMS) has now been approved for used in depressed patients resistant to drug therapy, and more is known about its mechanisms of action. In many patients, the severity of depression has been shown to correlate with the degree of decreased electrical activity in the prefrontal lobe. Treatment may result in marked improvement and even remission, but usually requires five 3. Vagal nerve stimulation, originally developed for treating certain types of seizure, has also been recently approved for drug resistant depression that has persisted for at least two years. It requires the surgical implantation of a pulse generator with leads to the left vagus nerve that is controlled externally. Like r. TMS it is expensive ($3. Non- invasive cranioelectrical stimulation (CES) provides similar benefits, and although approved products require a prescription in the U. S., they can be purchased directly in Canada and other countries for personal use. Alpha- Stim CES has been shown to improve depression 6. In addition, there have been no associated suicidal or homicidal ideation or significant adverse side effects after three decades of use, during which this and other devices have been progressively improved. Treatment protocols vary, but many depressed patients go into remission or experience significant benefits after 1. In addition to superior efficacy and safety, the one time price for the device is 2. SSRI therapy, and may be partially or completely reimbursable by insurance. Antidepressants often have to be taken perpetually, and can have serious withdrawal side effects, such as life threatening serotonin syndrome. It should be noted that Alpha- Stim CES has also been shown to be effective for treating anxiety, insomnia and other stress related complaints. However, it is crucial to avoid worthless copycat devices making similar claims supported only by lavish and self- serving testimonials, rather than studies published in peer reviewed scientific journals. The numerous difficulties associated with drugs have raised increasing concerns, since although the number of Americans on antidepressants doubled between 1. Antidepressants have now become the most commonly prescribed class of medications and are currently taken daily by one in ten people over the age of six. That figure is likely to escalate based on a paper in the current (January 2. Archives of General Psychiatry, which reported that only half of all Americans with depression receive treatment of any kind. And for those that do, only one in five is getting care in the form of medication and/or psychotherapy that conforms to American Psychiatric Association guidelines. Unfortunately, these recommendations date back to 2. FDA approved non drug therapies. The Association receives over thirty percent of its annual income from pharmaceutical manufacturers, and Congressional investigators have recently uncovered huge and hidden kickbacks to leading psychiatrists. As has happened in prior years, it is likely that those responsible for updating treatment recommendations could also have potential serious conflicts of interest . Health and Stress Newsletters #2 & 8 2. Fournier JC, De. Rubeis RJ, Hollon SD et al. Antidepressant drug effects and depression severity. JAMA, 2. 01. 0; 3. Sartorius A, Kiening KL, Kirsch P, von Gall CC, Haberkorn U, et al. Remission of major depression under deep brain stimulation of the lateral habenula in a therapy- refractory patient. Biol Psychiatry, 2. Jan 1. 5; 6. 7(2): e. Gilula, MF, Kirsch DL. Cranial electrotherapy stimulation review: A safer alternative to psychopharmaceuticals in the treatment of depression. Journal of Neurotherapy, 2. National patterns in antidepressant medication treatment. Arch Gen Psychiatry, 2. Depression care in the United States: Too little for too few. Arch Gen Psychiatry, 2. Cell Phone Stress, Depression, Fatigue and Saftey Scams. Concerns that cell phones might cause brain tumors have made headlines in recent months, sparked by speculations about Senator Ted Kennedy’s malignant glioma. Cell phones are also suspected to be responsible for an increased incidence of autism, ADHD, and other behavioral problems in children born to mothers who used them as little as two or three times a day during pregnancy. They absorb twice as much energy as adults and a two- minute call can alter brain function in a child for an hour. Many teenagers become addicted to their cell phones and suffer withdrawal symptoms if they are not immediately available. I am worried about the future of peoples’ wellness and how a newly recognized, yet rapidly emerging toxin called electropollution may be silently accelerating the aging process. It is dangerously increasing stress levels, preventing essential nutrients from entering the body’s cell, as well as keeping damaging toxins from exiting them. BIOPRO Technology is breaking new grounds, with the introduction of BIOPRO Cell Chips, Universal Chips, home harmoniser and Water appliances that will reduce the dangers from electromagnetic radiation emitted by cell phones, PDAs, Bluetooth handsets and other home appliances.”Electromagnetic radiation can only be blocked by dense materials like lead. Yet, they include the standard disclaimer that “These statements have not been evaluated by the FDA.
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. Archives
November 2017
Categories |